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       Semaglutide Correct Use
       By: Road2HardCoreIron Date: April 8, 2023, 7:52 am
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       (Dose varies Person)**********
       Semaglutide
       Description
       Semaglutide is a recombinant DNA produced polypeptide analogue
       of human glucagon-like peptide-1 (GLP-1) which is used in
       combination with diet and exercise in the therapy of type 2
       diabetes, either alone or in combination with other antidiabetic
       agents. There have been no published reports of hepatotoxicity
       attributed to semaglutide therapy.
       Semaglutide is a polypeptide that contains a linear sequence of
       31 amino acids joined together by peptide linkages. It is an
       agonist of glucagon-like peptide-1 receptors (GLP-1 AR) and used
       for the treatment of type 2 diabetes. It has a role as a
       hypoglycemic agent, a glucagon-like peptide-1 receptor agonist,
       an anti-obesity agent, a neuroprotective agent and an appetite
       depressant. It is a polypeptide and a lipopeptide.
       Obesity is a global health challenge with few pharmacologic
       options. Whether adults with obesity can achieve weight loss
       with once-weekly semaglutide at a dose of 2 mg as an adjunct to
       lifestyle intervention has not been confirmed.
       
       Semaglutide (Glucagon-Like Peptide-1) Overview
       Semaglutide, GLP-1, short for glucagon-like peptide-1 is a
       short, naturally occurring peptide hormone just 30-31 amino
       acids in length. Its primary physiologic function is to lower
       blood sugar levels by naturally enhancing insulin secretion. It
       also plays roles in protection beta cell insulin stores by
       promoting insulin gene transcription and has been linked with
       neurotrophic effects in the brain and central nervous system.
       In the GI system, GLP-1 has been shown to significantly decrease
       appetite by delaying gastric emptying and reducing intestinal
       motility. Preliminary research has shown impacts of GLP-1 in the
       heart, fat, muscles, bones, liver, lungs, and kidneys as well.
       The primary focus of GLP-1 research has been in the realm of
       diabetes treatment/prevention as well as appetite suppression.
       Secondary research focuses on the potential cardiovascular
       benefits of the peptide. More recent, and thus less robust,
       research focuses on the ability of GLP-1 to stave off
       neurodegenerative disease. Though this latter area of research
       is newest, it is also the fast-growing area of GLP-1 study now
       that the peptide has been revealed to slow or prevent the
       accumulation of amyloid beta plaques in the setting of
       Alzheimer’s disease.
       Semaglutide is a glucagon-like peptide-1 receptor agonist. It
       increases the production of insulin, a hormone that lowers the
       blood sugar level. It also appears to enhance growth of β
       cells in the pancreas, which are the sites of insulin
       production.
       In short, it inhibits glucagon, which is a hormone that
       increases blood sugar. It additionally reduces food intake by
       lowering appetite and slows down digestion in the stomach. In
       this way it reduces body fat.
       
       Semaglutide GLP-1 research
       The Incretin Effect of GLP-1
       Perhaps the most important effect that GLP-1 has, according to
       Dr. Holst, is referred to as the “incretin effect.” Incretins
       are a group of metabolic hormones, released by the GI tract,
       that cause a decrease in blood glucose (sugar) levels. GLP-1 has
       been shown to be one of the two most important hormones (the
       other being GIP) to stimulate the incretin effect in rodent
       models. Though GIP circulates at levels roughly 10 times higher
       than that of GLP-1, there is evidence that GLP-1 is the more
       potent of the two molecules, particularly when levels of blood
       glucose are quite high.
       A GLP-1 receptor has been identified on the surface of
       pancreatic beta cells, making it clear that GLP-1 directly
       stimulates the exocytosis of insulin from the pancreas. When
       combined with sulfonylurea drugs, GLP-1 has been shown to boost
       insulin secretion enough to cause mild hypoglycemia in up to 40%
       of subjects[1]. Of course, increased insulin secretion is
       associated with a number of trophic effects including increased
       protein synthesis, reduction in the breakdown of protein, and
       increased uptake of amino acids by skeletal muscle.
       
       GLP-1 and Beta Cell Protection
       Research in animal models suggests that GLP-1 can stimulate the
       growth and proliferation of pancreatic beta cells and that it
       may stimulate the differentiation of new beta cells form
       progenitors in the pancreatic duct epithelium. Research has also
       shown that GLP-1 inhibits beta cell apoptosis. Taken in sum,
       these effects tip the usual balance of beta cell growth and
       death toward growth, suggesting that the peptide may be useful
       in treating diabetes and in protecting the pancreas against
       insult that harms beta cells.
       In one particularly compelling trial, GLP-1 was shown to inhibit
       the death of beta cells caused by enhanced levels of
       inflammatory cytokines. In fact, mouse models of type 1 diabetes
       have revealed that GLP-1 protects islet cells from destruction
       and may, in fact, be a useful means of preventing onset of the
       type 1 diabetes.
       
       GLP-1 and Appetite
       Research in mouse models suggests that administration of GLP-1,
       and its similar cousin GLP-1, into the brains of mice can reduce
       the drive to eat and inhibit food intake. It appears that GLP-1
       may actually enhance feelings of satiety, helping individuals to
       feel fuller and reducing hunger indirectly. Recent clinical
       studies have shown in mice that twice daily administration of
       GLP-1 receptor agonists cause gradual, linear weight loss. Over
       a long period, this weight loss is associated with significant
       improvement in cardiovascular risk factors and a reduction in
       hemoglobin A1C levels, the latter of these being a proxy marker
       for the severity of diabetes and the quality of blood sugar
       control attained via treatment.
       
       Potential Cardiovascular Benefits of GLP-1
       It is now know that GLP-1 receptors are distributed throughout
       the heart and act to improve cardiac function in certain
       settings by boosting heart rate and reducing left ventricular
       end-diastolic pressure. The latter may not seem like much, but
       increased LV end-diastolic pressure is associated with LV
       hypertrophy, cardiac remodeling, and eventual heart failure.
       Recent evidence has even suggested that GLP-1 could play role in
       decreasing the overall damaged caused by a heart attack. It
       appears that the peptide improves cardiac muscle glucose uptake,
       thereby helping struggling ischemic heart muscle cells to get
       the nutrition they need to continue functioning and avoid
       programmed cell death. The increase in glucose uptake in these
       cells appears to independent of insulin.
       Large infusions of GLP-1 into dogs have been shown to improve LV
       performance and reduce systemic vascular resistance. The latter
       effect can help to reduce blood pressure and ease strain on the
       heart as a result. This, in turn, can help to reduce the
       long-term consequences of high blood pressure such as LV
       remodeling, vascular thickening, and heart failure. According to
       Dr. Holst, administration of GLP-1 following cardiac injury has
       “constantly increased myocardial performance both in
       experimental animal models and in patients.”
       
       GLP-1 and the Brain
       There is some evidence to suggest that GLP-1 can improve
       learning and help to protect neurons against neurodegenerative
       diseases such as Alzheimer’s disease. In one study, GLP-1 was
       shown to enhance associative and spatial learning in mice and
       even to improve learning deficits in mice with specific gene
       defects. In rats that over-express the GLP-1 receptor in certain
       regions of the brain, learning and memory are both significantly
       better than in their normal controls.
       Additional research in mice has shown that GLP-1 can help to
       protect against excitotoxic neuron damage, completely protecting
       rat models of neurodegeneration against glutamate-induced
       apoptosis. The peptide can even stimulate neurite outgrowth in
       cultured cells. Researchers are hopeful that additional research
       on GLP-1 will reveal how it might be used to halt or reverse
       certain neurodegenerative diseases.
       Interestingly, GLP-1 and its analogue exendin-4 have been shown
       in mouse models to reduce levels of amyloid-beta in the brain as
       well as the beta-amyloid precursor protein found in neurons.
       Amyloid beta is the primary component of the plaques observed in
       Alzheimer’s disease, plaques which, while not necessarily known
       to be causative, are associated with the severity of the
       disease. It remains to be seen if preventing amyloid beta
       accumulation can protect against the effects of Alzheimer’s
       disease, but this research is, at the very least, a tantalizing
       clue as to how scientists my intervene in the progression of
       mild cognitive impairment to full Alzheimer’s disease.
       GLP-1 exhibits minimal to moderate side effects, low oral and
       excellent subcutaneous bioavailability in mice. Per kg dosage in
       mice does not scale to humans.
       
       
       Semaglutide Questions
       • How to use semaglutide for weight loss?
       Taking 2.4 Mg/0.75 Ml Subcutaneous Pen Injector as example,
       inject this medication under the skin in the thigh, abdomen, or
       upper arm as directed by your doctor, usually once weekly. It
       may be used with or without food. The dosage is based on your
       medical condition and response to treatment. Your doctor will
       start you on a low dose first to decrease your risk of
       stomach/abdominal side effects, and gradually increase your
       dose. Follow your doctor’s instructions carefully.
       Before using, check this product visually for particles or
       discoloration. If either is present, do not use the liquid.
       Before injecting each dose, clean the injection site with
       rubbing alcohol. Change the injection site each time to lessen
       injury under the skin. Do not inject in an area that is tender,
       bruised, red, hard, or has scars or stretch marks.
       Use this medication regularly to get the most benefit from it.
       To help you remember, use it on the same day and time each week.
       It may help to mark your calendar with a reminder. Carefully
       follow the meal plan and exercise program your doctor has
       recommended. Learn how to store and discard medical supplies
       safely.
       
       • How does semaglutide burn fat?
       Patients can lose significant weight on semaglutide because it
       suppresses appetite. Semaglutide can be taken orally or by
       injection, according to Novo Nordisk, and it works by increasing
       the production of insulin, a hormone produced by the pancreas
       that helps regulate blood sugar.
       
       What are the side effects of semaglutide?
       Semaglutide injection may cause side effects. Tell your doctor
       if any of these symptoms are severe or do not go away: nausea.
       vomiting. diarrhea. abdominal pain. constipation. heartburn.
       burping.
       
       Does semaglutide cause hair loss?
       No, hair loss wasn’t reported as a side effect by people taking
       Ozempic in clinical studies. But hair loss and hair thinning may
       occur in people who have diabetes and consistently high blood
       sugar levels. If you have questions about hair loss, talk with
       your doctor or pharmacist.
       
       How quickly do you lose weight with semaglutide?
       Despite the promising research, there’s one major caveat to the
       medication: in order to keep the weight off, patients need to
       continue taking it. One recent study showed that patients on
       semaglutide lost 10% of their body weight in 20 weeks, but
       regained nearly all of it after the treatment.
       
       How long do you stay on semaglutide?
       Most patients will start semaglutide treatment at the lower 0.25
       mg dose injected once a week for 4 weeks. The 0.25 mg dose is
       not used as your final maintenance dose to lower your blood
       sugar.
       
       Where do you inject semaglutide?
       If you will be using semaglutide at home, your doctor will teach
       you how the injections will be given. Be sure you understand
       exactly how the medicine is to be injected. This medicine is
       given as a shot under the skin of your stomach, thighs, or upper
       arm. Use a different body area each time you give yourself a
       shot.
       
       Does semaglutide make tired?
       Swelling/redness/itching at the injection site, tiredness,
       nausea, vomiting, diarrhea, or constipation may occur. Nausea
       usually lessens as you continue to use semaglutide. If any of
       these effects last or get worse, tell your doctor or pharmacist
       promptly. Other adverse reactions with a frequency of >0.4% were
       associated with OZEMPIC include fatigue, dysgeusia and
       dizziness.
       
       What happens when you stop semaglutide?
       Continuation of semaglutide after 20 weeks of initial therapy
       leads to significant continued weight loss, according to a new
       study, but stopping the therapy causes patients to regain much
       of the weight they initially lost.
       
       Is semaglutide safe long term?
       The study demonstrates that long-term use of oral semaglutide
       with flexible dose adjustment results in durable improvements in
       glycemic control and further reductions in body weight and is
       generally well tolerated.
       
       
       
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