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   DIR Return to: TECFIDERA (dimethyl fumarate, BG-12, Fumaderm)
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       #Post#: 516--------------------------------------------------
       Dimethyl fumarate for RRMS (Lancet Neurology)
       By: agate Date: October 14, 2014, 6:11 pm
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       From Lancet Neurology, November 2014:
       [quote]Dimethyl fumarate for relapsing-remitting multiple
       sclerosis
       Martyn J Burke a, Joanna Richardson a, Elisabeth George a,
       Amanda I Adler a
       On August 27, 2014, the National Institute for Health and Care
       Excellence (NICE) published guidance recommending dimethyl
       fumarate as an option for the treatment of active
       relapsing-remitting multiple sclerosis (RRMS) that is neither
       highly active nor rapidly evolving severe RRMS.
       NICE appraised dimethyl fumarate under the Single Technology
       Appraisal process. Biogen Idec, the manufacturer of dimethyl
       fumarate, under the brand name Tecfidera, submitted clinical
       evidence and a health-economics model, which were critiqued by
       an independent Evidence Review Group from the University of
       York. Furthermore, an independent NICE Appraisal Committee met
       twice on August 21, 2013 and May 21, 2014 to develop guidance on
       the use of dimethyl fumarate. Clinical specialists and a patient
       expert attended the first meeting, and Biogen Idec
       representatives attended both meetings.
       The clinical evidence focused on two randomised controlled
       trials, DEFINE and CONFIRM. In DEFINE, 1237 adults with RRMS
       were randomly assigned to 240 mg dimethyl fumarate taken orally,
       twice daily (n=410) or three times daily (n=416), or to placebo
       (n=408). In CONFIRM, 1430 adults with RRMS were randomly
       assigned to 240 mg dimethyl fumarate twice daily (n=359) or
       three times daily (n=345), to 20 mg glatiramer acetate once
       daily (n=350; open-label), or to placebo (n=363). In both
       trials, patients were treated for up to 96 weeks. Patients
       stopped treatment if they did not tolerate the study drug or
       withdrew consent.
       The primary outcome measure was the proportion of patients with
       a relapse at 2 years in DEFINE, and the annualised relapse rate
       at 2 years in CONFIRM. In DEFINE, the proportion of patients
       with a relapse was lower in the group treated with dimethyl
       fumarate than in the group that was given placebo (27% vs 46%;
       hazard ratio [HR] 0·51, 95% CI 0·40—0·66). In CONFIRM, the
       annualised relapse rate was 0·22 in patients treated with
       dimethyl fumarate and 0·40 in patients given placebo (relative
       risk 0·56, 95% CI 0·42—0·74).
       Secondary outcomes in both trials included progression of
       disability on the Expanded Disability Status Scale (EDSS) from 0
       to 10, with high scores indicating high levels of disability.
       Patients in DEFINE who were randomly assigned to dimethyl
       fumarate were less likely to have disability progression
       sustained for 3 months than were patients randomly assigned to
       placebo (16% vs 27%; HR 0·62, 95% CI 0·44—0·87). In CONFIRM, any
       benefit in terms of disability progression was not significant
       (13% vs 17%; HR 0·79, 95% CI 0·52—1·19). Biogen Idec also
       analysed disability progression sustained for 6 months in
       patients given dimethyl fumarate or placebo in DEFINE (HR 0·77,
       95% CI 0·52—1·14) and CONFIRM (HR 0·62, 95% CI 0·37—1·03).
       Although DEFINE and CONFIRM both included MRI assessments of the
       brain, these outcomes were not included in the appraisal because
       stakeholders agreed during the scoping stage at the start of the
       appraisal that the outcomes of primary importance to patients
       with RRMS were relapse rate, relapse severity, and disability.
       Biogen Idec did a mixed treatment comparison unadjusted for
       relapse rates to estimate the effectiveness of dimethyl fumarate
       compared with disease-modifying therapies including beta
       interferon-1a (Avonex, Rebif-22, and Rebif-44), beta
       interferon-1b (Betaferon), glatiramer acetate, fingolimod,
       natalizumab, and placebo in people with active RRMS. Biogen
       Idec's mixed treatment comparison showed the annualised relapse
       rate and proportion of patients with relapses were significantly
       lower in patients treated with dimethyl fumarate than patients
       given placebo, glatiramer acetate, or all beta interferon
       treatments, and disability progression sustained for 3 months
       was decreased in patients treated with dimethyl fumarate
       compared with placebo. Biogen Idec did not estimate the
       effectiveness of dimethyl fumarate in highly active or rapidly
       evolving severe RRMS, stating that most trials included in its
       mixed treatment comparison excluded these subgroups.
       To determine the cost effectiveness of dimethyl fumarate
       compared with other disease-modifying therapies, Biogen Idec
       used a Markov model to estimate disease progression through 21
       health states defined by EDSS, progression to secondary
       progressive multiple sclerosis, and death. In each cycle, a
       patient with RRMS could move to a lower or higher score on EDSS,
       remain stable, or could progress from RRMS to secondary
       progressive multiple sclerosis. Biogen Idec assumed, in the
       absence of evidence to the contrary, that the treatment effect
       of dimethyl fumarate decreases over time.
       During its first meeting, the Committee heard that most patients
       with active disease, defined as two relapses in the previous 2
       years, would be offered a disease-modifying therapy in current
       clinical practice. Based on the evidence, the Committee
       concluded that dimethyl fumarate decreases relapses compared
       with placebo in patients with RRMS. The Committee understood
       from clinical specialists that although most trials do not
       assess disability progression sustained for 6 months the
       assessment of disability progression sustained for 3 months
       provides a more robust indication of the treatment effect given
       that patients may recover from relapse. The Committee recognised
       that to model the natural history of RRMS with low EDSS scores,
       Biogen Idec used placebo data from DEFINE and CONFIRM, which
       reflected the population in UK clinical practice more closely
       than the population in the older London Ontario data used in
       previous NICE appraisals of treatments for RRMS. However, the
       Committee agreed that uncertainty remained in estimates of the
       incremental cost-effectiveness ratios. The Committee was
       concerned about the economic model's prediction that the sooner
       a patient stops treatment, the more cost effective the
       treatment. The Committee, therefore, requested that Biogen Idec
       clarify several issues and undertake further analyses. Among
       other requests, the Committee asked Biogen Idec to revise the
       probabilistic incremental cost-effectiveness ratios
       incorporating mixed treatment comparison results adjusted for
       relapse rates of patients entering the trials, to revise
       resource use and cost estimates, and exclude non-health-care
       costs. The Committee also requested that Biogen Idec externally
       validate its incremental cost-effectiveness ratio and compare
       estimates of cost-effectiveness of current treatments with
       treatments in the NHS risk-sharing scheme for multiple sclerosis
       (a scheme to ensure that patients with multiple sclerosis can
       access Avonex, Betaferon, Copaxone, and Rebif-22).
       At the second meeting, the Committee considered the additional
       analyses from Biogen Idec, a critique of the additional analyses
       from the independent Evidence Review Group; and responses to the
       Committee's preliminary guidance from consultees, commentators,
       and the general public. The Committee noted that uncertainty
       remained about whether Biogen Idec had adjusted appropriately
       for the relapse rate of patients entering the trials, and
       therefore, agreed that the unadjusted mixed treatment comparison
       was preferable because it provided a better statistical fit. The
       Committee concluded that dimethyl fumarate decreases relapse
       rates more effectively than beta interferons or glatiramer
       acetate, and decreases the progression of disability as
       effectively as beta interferons or glatiramer acetate. The
       Committee noted that it had insufficient evidence to recommend
       dimethyl fumarate for people with highly active or rapidly
       evolving severe RRMS, for whom fingolimod and natalizumab,
       respectively, have been recommended by NICE.
       The Committee was satisfied that the Biogen Idec economic model
       was robust for decision-making after considering the results
       from the external validation. The Committee noted that Biogen
       Idec also provided incremental cost-effectiveness ratios for a
       scenario using its preferred assumptions, in which it
       incorporated estimates from the unadjusted mixed treatment
       comparison and monitoring requirements based on its summary of
       product characteristics. The Committee acknowledged that Biogen
       Idec had appropriately modelled health-related quality of life
       using EQ-5D (a measure of health outcome) data and had included
       a decrease in treatment effect over time. The Committee
       acknowledged that Rebif-22 appeared to be the most
       cost-effective comparator, but that Rebif-22 is a step-down
       therapy for patients who cannot tolerate the recommended high
       dose (ie, Rebif-44). The Committee, therefore, disregarded
       Rebif-22, and based the most plausible incremental
       cost-effectiveness ratio on a comparison of dimethyl fumarate
       with glatiramer acetate (the next most cost-effective comparator
       after Rebif-22) using Biogen Idec's preferred model. This
       analysis showed an incremental cost-effectiveness ratio of about
       £27 700 per quality-adjusted life year (QALY) gained based on
       drug costs including a patient access scheme agreed with the
       Department of Health, UK. The Committee agreed that if Biogen
       Idec's model had overestimated the drug's decreased treatment
       effect over time, and had included the non-medical costs covered
       by Personal Social Services, the incremental cost-effectiveness
       ratio of dimethyl fumarate compared with glatiramer acetate
       would decrease. The Committee noted that benefits not reflected
       in QALYs, such as taking dimethyl fumarate orally compared with
       injections of glatiramer acetate, and its shorter washout
       period, could further decrease the incremental
       cost-effectiveness ratio. In conclusion, the Committee deemed
       dimethyl fumarate to be a cost-effective use of NHS resources in
       the treatment of adults who have active RRMS, but who have
       neither highly active nor rapidly evolving severe RRMS, only if
       the manufacturer provides dimethyl fumarate with the discount
       agreed in the patient access scheme.
       NICE issued a final appraisal determination stating the
       Committee's decision. NICE offered stakeholders the opportunity
       to appeal against the recommendations, but no appeals were
       received.
       __________________
       We declare no competing interests.
       a National Institute for Health and Care Excellence, City Tower,
       Piccadilly Plaza, Manchester, M1 4BT, UK[/quote]
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